MULTI SCIENCES
Human TWEAK Antibody ELISA Kit
Human TWEAK Antibody ELISA Kit
SKU:EK1256
Product Details
Factory Name | MultiSciences |
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Product Code | 70-EK1256 |
CatNum | EK1256 |
Product Name | Human TWEAK ELISA Kit |
Customs Name | Customs Name |
Type | ELISA Kit |
Application | ELISA |
Reactivity | Human |
SDS | SDS - EK1256 |
Assay Type | Sandwich ELISA |
Suitable Sample Type | serum, plasma, cell culture supernates |
Format | 96-well strip plate |
Storage | 4℃ (unopened)standard stored at -20℃, others stored at 4℃ (opened) |
Shipping Condition | 4℃ |
Sample Volume | 50 μl |
Sensitivity | 0.87 pg/ml |
Standard Curve Range | 15.63- 1000 pg/ml |
Spike Recovery Range | 88% - 117% |
Mean Spike Recovery | 1.05 |
CV of Intra plate | 3.7% - 6.6% |
CV of Inter plate | 4.7% - 6.5% |
Components | 96-well polystyrene microplate coated with a monoclonal antibody against TWEAK Human TWEAK Standard, lyophilized TWEAK Detect Antibody Standard Diluent Assay Buffer (10×) Substrate (TMB) Stop Solution Washing Buffer (20×) Plate Covers |
Describtion | This assay employs the quantitative sandwich enzyme immunoassay technique for the quantitative detection of human TWEAK.The Human TWEAK ELISA is for research use only. Not for diagnostic or therapeutic procedures. The Human IGFBP-1 ELISA is for research use only. Not for diagnostic or therapeutic procedures. Tumor necrosis factor-like weak inducer of apoptosis (TWEAK) was discovered in 1997. The protein encoded by this gene is a cytokine that belongs to the tumor necrosis factor (TNF) ligand family. Leukocytes are the main source of TWEAK including human resting and activated monocytes, dendritic cells and natural killer cells. TWEAK can induce apoptosis via multiple pathways of cell death in a cell type-specific manner. This cytokine is also found to promote proliferation and migration of endothelial cells, and thus acts as a regulator of angiogenesis. Excessive activation of the TWEAK pathway in chronic injury has been described to promote pathological tissue changes including chronic inflammation, fibrosis and angiogenesis. |
