Skip to product information
1 of 1

MULTI SCIENCES

Rat IL-1beta Cytokine ELISA Kit

Rat IL-1beta Cytokine ELISA Kit

SKU:EK301B

Regular price Send Inquiry
Regular price Sale price Send Inquiry
Sale Sold out
View full details

Product Details

Rat IL-1beta Cytokine ELISA Kit

Brand MultiSciences
CatNum 70-EK301B
Product Name Rat IL-1beta ELISA Kit
Customs Name Rat IL-1beta ELISA Kit
Application ELISA
Reactivity Rat
Assay Type Sandwich ELISA
Suitable Sample Type serum, plasma, cell culture supernates
Format 96-well strip plate
Storage 4℃ (unopened) standard stored at -20℃, others stored at 4℃ (opened)
Shipping Condition 4℃
Sample Volume 20 μl
Sensitivity 1.42 pg/ml
Standard Curve Range 31.25 - 2000 pg/ml
Spike Recovery Range 84 % - 110 %
Mean Spike Recovery 1.02
CV of Intra plate 5.0 % - 5.1 %
CV of Inter plate 5.5 % - 6.7 %
Release Date 2016/8/20
Components 96-well polystyrene microplate coated with a monoclonal antibody against IL-1β
Rat IL-1β Standard, lyophilized
IL-1β Detect Antibody
Standard Diluent
Streptavidin-HRP
Assay Buffer (10×)
Substrate (TMB)
Stop Solution
washing Buffer (20×)
Plate Covers
Describtion This assay employs the quantitative sandwich enzyme immunoassay technique for the quantitative detection of rat IL-1β. The Rat IL-1β ELISA is for research use only. Not for diagnostic or therapeutic procedures.
Interleukin 1 (IL-1) is an extracellular peptide of 17 kDa that designates two proteins, IL-1α and IL-1β. IL-1β is produced by activated macrophages as a proprotein, which is proteolytically processed to its active form by caspase 1 (CASP1/ICE). This cytokine is an important mediator of the inflammatory response, and is involved in a variety of cellular activities, including cell proliferation, differentiation, and apoptosis.The interleukin-1 (IL-1) species represent an important family of biologically active mono nuclear cell-derived proteins which are involved in inflammatory reactions and in immune responses.
Increased production of IL-1β causes a number of different autoinflammatory syndromes, most notably the monogenic conditions referred to as CAPS, due to mutations in the inflammasome receptor NLRP3 which triggers processing of IL-1β.